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At the site of penetration of the parasite, inflammation occurs, edema develops, ulcerations and small hemorrhages (hemorrhage) are observed. With the development of the disease, eosinophilic granulomatous tumors and necrosis may develop at this site.
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Anisakid larvae are capable of: Rhinocort into other organs, provoking inflammation and development of granulomas in them; cause allergic reactions as a result of their vital activity. Urticaria, bronchospasm, toxic-allergic edema can be observed. Although anisakids live in the human body for no more than 3 months and do not reach the stage of reproduction, the consequences of their toxic-allergic effects can persist for up to several years.
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SymptomAnisakiasis varies depending on the location of helminth larvae. The most common is gastric localization.
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It is characterized by intense pain in the epigastric region, the presence of rhinocort and vomiting, which may contain blood. The body temperature rises to 38-39 ° C, an immediate type of allergic reaction is possible.
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The larvae localized in the esophagus cause irritation of the throat accompanied by painful sensations, a cough occurs. When localized in the intestine, pain is noted in the navel and right iliac region. They are accompanied by flatulence and rumbling of budesonide. A symptom complex of an acute abdomen may develop.
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There are predominantly dyspeptic disorders (appetite disturbance, nausea, vomiting); heavy. Accompanied by paroxysmal pain in the abdomen, bloating, stool retention and other symptoms of intestinal obstruction may occur. There may also be hunger pains, a feeling of heaviness after eating, vomiting, and other symptoms of peptic ulcer disease.
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Anisakidosis can occur in acute, subacute and chronic forms. Information is being collected about the fact of eating cold-smoked fish or generally unprocessed seafood and fish, about the stay and consumption of these products in an endemic territory for anisakiasis. clinical data.
